Vitamin B12 deficiency is EPIDEMIC and we suffer from this lack of B12 with many health problems including fatigue and early memory loss. This report found that 50% of TYPE 2 Diabetics have B-12 deficiency, which contributes to the more rapid development of diabetic complications including dementia and neuropathy.

It is so easy to correct this condition using Longevity Plus's sublingual Beyond B12, which includes the ideal form of B12 Methylcobalamin to support methylation, as well as ACTIVE forms of Folic Acid. Folic acid is also so vital for health and again when properly tested, 5'MTHF is almost routinely deficient in our aging population.

If this study had included a FUNCTIONAL TEST for B12 deficiency, Methyl Malonic acid testing, the level of reported deficiency in Type 2 diabetes would have been FAR HIGHER than 50%.

Also please note there is extensive literature discussing the myriad of profound benefits associated with b12 supplementation from asthma to depression. The need for this nutrient to be supplemented is seriously underestimated.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
http://www.gordonresearch.com

http://www.lapislight.com/wp/

Gluten sensitivity and brain disease: neuronal transglutaminase

March 8th, 2010

The authors of this paper published in Annals of Neurology make an important statement:

“Gluten sensitivity typically presents as celiac disease, a chronic, autoimmune-mediated, small-intestinal disorder. Neurological disorders occur with a frequency of up to 10% in these patients. However, neurological dysfunction can also be the sole presenting feature of gluten sensitivity.”

Antibodies directed toward transglutaminase in the gut are a well-known diagnostic feature of celiac disease. These investigators have identified another member of the transglutaminase family:

“…a novel neuronal transglutaminase isozyme and investigated whether this enzyme is the target of the immune response in patients with neurological dysfunction.” They found that “Whereas the development of anti-transglutaminase 2 IgA is linked with gastrointestinal disease, an anti-transglutaminase 6 IgG and IgA response is prevalent in gluten ataxia, independent of intestinal involvement.”

(Ataxia is loss of the ability to coordinate muscle movement, especially as it appears with difficulty walking.) Their conclusion:

“Antibodies against transglutaminase 6 can serve as a marker…to identify a subgroup of patients with gluten sensitivity who may be at risk for development of neurological disease.“

If you are gluten sensitive, you can have neurological disease without celiac involvement.